95 Dr. Aga-Mizrachi Shlomit Telephone: 077-2343808 Cell phone: 0545240974 Email: Shlomit.Mizrachi@gmail.com Google Scholar: https://scholar.google.com/citations?user=_8I9XbkAAAAJ&hl=iw&oi=ao Our research in the Behavioral Neuroscience lab focused on attention and on the ability to perform social-cooperation. By combining behavioural knowledge and technological tools, we developed fully automated maze, which enables a thorough examination of the ability to perform social cooperation in a rodent model. We are intensively trying to decipher the underlying mechanism of the ability to socially cooperate and we provide for the first-time evidence for the inheritability of social regulation/dysregulation, deciphering the underlying mechanism in various aspects such as behavioral, anatomical (MRI), neural plasticity, proteomic and pharmacology. Peer-Reviewed Papers in Refereed Journals 1. Rosenzeieg, T*. Aga-Mizrachi, S., Bak, A., and Sampson, S.R. Src Tyrosine Kinase Regulates Insulin-Induced Activation of Protein Kinase C (PKC) in Skeletal Muscle. Cell Signal. 2004; 16:1299-1308. (IF: 4.305, Q1) (*equal contribution) 2. Cipok M., Aga-Mizrachi S., Bak A., Feurstein T., Steinhart R., Brodie C. and Sampson, S.R. Protein kinase C alpha Regulates Insulin Receptor Signaling in Skeletal Muscle. Biochem Biophys Res Commun. 2006; 345 (2): 817-824. (IF: 2.648, Q1) 3. Aga-Mizrachi S, Brutman-Barazani T, Jacob AI, Bak A, Elson A, Sampson SR. Cytosolic protein tyrosine phosphatase-epsilon is a negative regulator of insulin signaling in skeletal muscle. Endocrinology. 2008; 149(2):605-14. (IF: 4.276, Q1) 4. Jacob A, Horovitz-Fried M, Aga-Mizrachi S, Brutman-Barazani T, Okhrimenko H, Zick Y, Brodie C, Samspon S. The regulatory domain of protein kinase C delta positively regulates insulin receptor signaling. J Mol Endocrinol. 2010; 44(3):155-69. (IF: 3.081, Q1) 5. Oshrat Dadush*, Shlomit Aga-Mizrachi*, Keren Ettinger, Rinat Tabackman, Moran Elbaz, Yakov Fellig, Vivian Barak and Yoram Nevo. Improved muscle strength and mobility in Glatiramer acetate treated dy2J/dy2J mice model of congenital muscular dystrophy. Neuromuscul Disord. 2010; 20(4):267-72.) (IF: 2.735, Q2) (*equal contribution) 6. Nevo Y*, Aga-Mizrachi S*, Elmakayes E, Yanay N, Ettinger K, Elbaz M, Brunschwig Z, Dadush O, Elad-Sfadia G, Haklai R, Kloog Y, Chapman J, Reif S. The Ras Antagonist, Farnesylthiosalicylic Acid (FTS), Decreases Fibrosis and Improves Muscle Strength in dy/dy Mouse Model of Muscular Dystrophy. PLoS One. 2011; 22; 6(3):e18049. (IF: 4.092, Q1) (*equal contribution) 7. Avital A, Dolev T, Aga-Mizrachi S, Zubedat S. Environmental Enrichment Preceding Early Adulthood Methylphenidate Treatment Leads to Long Term Increase of Corticosterone and Testosterone in the Rat. PLoS One. 2011; 6(7):e22059. (IF: 4.092, Q1)
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